A potential diagnostic biomarker: Proteasome LMP2/b1i-differential expression in human uterus neoplasm

نویسندگان

  • Takuma Hayashi
  • Akiko Horiuchi
  • Hiroyuki Aburatani
  • Nobuo Yaegashi
  • Ikuo Konishi
چکیده

1 Dept. of Immunology and Infectious Disease, Shinshu University Graduate School of Medicine, Matsumoto-city, Nagano 390-8621, Japan, 2 Dept. of Obstetrics and Gynecology, Shinshu University School of Medicine, Matsumoto-city, Nagano 390-8621, Japan. 3 The Cancer System Laboratory, Research Center for Advanced Science and Technology, The University of Tokyo, Tokyo 153-9804 Japan. 4 Dept. of Obstetrics and Gynecology, Tohoku University Graduate School of Medicine, Miyagi 980-8574 Japan. 5 Picower Institution and Dept. of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139-4307 USA. 6 Dept. of Obstetrics and Gynecology, Kyoto University Graduate School of Medicine, Kyoto-city, Kyoto 606-8507, Japan. 7 Promoting Business using Advanced Technology, Japan Science and Technology Agency (JST), Chiyoda-ku, Tokyo 102-8666, Japan.

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

A novel diagnostic marker: Proteasome LMP2/β1i-differential expression in human uterus mesenchymal tumors

Uterine leiomyosarcoma (LMS) develops more often in the muscle tissue layer of the uterine body than in the uterine cervix. The development of gynecologic tumors is often correlated with female hormone secretion; however, the development of uterine LMS is not substantially correlated with hormonal conditions, and the risk factors are not yet known. Importantly, a diagnostic-biomarker, which dis...

متن کامل

Molecular Approach to Uterine Leiomyosarcoma: LMP2-Deficient Mice as an Animal Model of Spontaneous Uterine Leiomyosarcoma

Uterine leiomyosarcoma (LMS) develops more often in the muscle tissue layer of the uterine body than in the uterine cervix. The development of gynecologic tumors is often correlated with female hormone secretion; however, the development of uterine LMS is not substantially correlated with hormonal conditions, and the risk factors are not yet known. Importantly, a diagnostic-biomarker which dist...

متن کامل

Candidate Molecules and ki-67/MIB1 as Novel Diagnostic Biomarker for Human Uterine Mesenchymal Tumors

Human uterine leiomyosarcoma (LMS) develops more often in the muscle tissue layer of the uterine body than in the uterine cervix. The development of gynecologic tumors is often correlated with female hormone secretion; however, the development of uterine LMS is not substantially correlated with hormonal conditions, and the risk factors are not yet known. Importantly, a diagnostic-biomarker, whi...

متن کامل

Molecular Approach to Uterine Leiomyosarcoma: LMP2- Deficient Mice as an Animal Model of Spontaneous Uterine Leiomyosarcoma Citation

The MIT Faculty has made this article openly available. Please share how this access benefits you. Your story matters. License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Uterine leiomyosarcoma (LMS) develops more often in the muscle tissue layer of the uterine body than in the uterine cervix. The development of gy...

متن کامل

Candidate Molecules as Tumor Suppressor for Human Uterine Mesenchymal Tumor

Uterine leiomyosarcoma (Ut-LMS) develops more often in myometrium of the uterine body than in the uterine cervix. The development of gynecologic tumors is often correlated with female hormone secretion; however, the development of Ut-LMS is not substantially correlated with hormonal conditions, and the risk factor(s) are not yet known. Importantly, a diagnostic-biomarker, which distinguishes ma...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:

دوره   شماره 

صفحات  -

تاریخ انتشار 2012